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Many Voices, One Freedom: United in the 1st Amendment

March 28, 2024

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As an 84-year-old cancer survivor who continues to interact face-to-face with my graduate students, I also have a known genetic family propensity for COVID vaccine-induced blood clotting. In addition, I recently contracted and recovered from omicron following two vax jabs received before I knew about that added clotting risk.

So, herein lies a question: Might omicron actually be a blessing — not just for my family and me, but also for many millions of others — perhaps serving as nature’s vaccine?

That intriguing possibility, but by no means any certainty, seems like a bright ray of hope in dark times of unrelenting disaster and dread vaccine-or-death mantra.

Having come to thoroughly distrust the Biden administration’s repeated misinformation on the subject — fundamentally including our own government’s NIH-confirmed role in sponsoring the gain-of-function experiments at the Chinese Wuhan laboratory that likely created the entire mess — I’ll share some insights gleaned from some other seemingly more agenda-independent sources.

A credibly cautious article posted in The Atlantic, “Should I Just Get Omicron Over With?” warns that whereas if you’re already vaccinated, an infection “might not make you super sick, but don’t count on it making you super immune either.”

The logic here is that since additional exposures to extra bits of a virus do seem to tend to build immunity incrementally, it’s “not irrational to imagine that an infection will leave one’s antiviral armor just a shade thicker.”

Analogously, the latest omicron wave has created a kind of post-COVID honeymoon phase, which like any honeymoon, is transient. In this regard, no combinations of vaccines or viruses can confer invulnerability to future COVID strains.

Whether acquired from an injection or an infection, immunity will always work in degrees, not absolutes.

Immunity is, in many ways, a game of repetition. The more frequently, and more intensely, immune cells are exposed to a threat, the more resolutely they’ll commit to fighting it, and the longer they’ll store away any microbial information they glean.

Over time, as new viral mutations pare down those protections, additional vaccine requirements and sickness can build them back up.

As Ai-ris Yonekura Collier, a physician and vaccine researcher at Beth Israel Deaconess Medical Center, explains, an infection on top of layered vaccinations can coax out “almost what you would call a boosted response.”

Together, vaccines and infections can confer what some immunologists refer to as “hybrid immunity,” potentially the best protection possible.

In theory, post-vaccination infections such as a highly mutated omicron may also influence immunity in beneficial ways vaccines can’t by offering more up-to-date protections as well as providing the body more complete information about the virus anatomy to help identify and destroy it, thus broadening the defense.

After nearly two years into this pandemic, with the U.S. reporting a record high of more than 1 million new COVID-19 cases earlier this month, many are wondering when a sufficient number of the population will reach a level of “herd immunity” so that the virus can no longer find a place to reproduce.

Dr. Luis Ostrosky-Zeichner, chief of infectious diseases with McGovern Medical School at the University of Texas Health Science Center at Houston, believes this may be possible with this omicron wave, and Israel’s top health adviser, Dr. Nachman Ash, agrees that the current surge might also make this happen in his country.

According to the Mayo Clinic, approximately 94% of people must be immune in order to successfully halt transmission.

Although many health experts say the best way to achieve some level of immunity is through vaccinations, those protections may not last or may become obsolete, just as with natural immunity.

So long as the virus is able to mutate, lasting immunity is likely off the table when another variant surges that can evade antibodies people have built up.

The eternal hope is that, like omicron, although highly infectious, the worst effects of future COVID variants can be mitigated through regularly adapted vaccines as the virus goes “endemic” over time with conventionally treatable symptoms.

Influenza, a respiratory virus commonly known as the flu, is an example, which a century after first appearing in 1918, still sees seasonal outbreaks requiring shots. Similarly, generations one hundred years from now may still be receiving periodic COVID shots.

So, getting back to the basic question I started with, the answer is “Yes,” it seems that omicron may turn out to act much like a natural vaccine against more dangerous previous variants — at least for now.

Nevertheless — as with all “vaccines” — we can’t necessarily count it to offer protections against its own — or a cousin’s — future mutant progeny.

Whereas the Trump administration’s Operation Warp Speed evidenced remarkably rapid progress in developing mRNA vaccine platform technologies, omicron wasted no time leapfrogging ahead of the defense.

In addition to safer time-proven vaccines, there’s also a critical need for effective new therapeutics to build our body’s ability to offset and combat infections, particularly at early and most treatable stages.

Each of us as well has vital self-defense roles through healthy lifestyles: eat right, drop some weight if you need to, remain physically and mentally active, and practice good hygiene.

And above all, since we apparently can’t entirely count on herd immunity for salvation, let’s at least take this experience as a dire warning — perhaps our last — not to bioengineer viruses that readily outsmart the mutant strain of herd imbecility responsible for creating them in the first place.

MANY VOICES, ONE FREEDOM: UNITED IN THE 1ST AMENDMENT

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Catherine L Currier
Catherine L Currier
2 years ago

No, influenza doesn’t “require” any shot whatsoever! #vaccinefree only way to be 😁

p. gutierrez
p. gutierrez
2 years ago

Some excerpts from better preprints for Omicron.

Glocker study group thinks that Omicron may enhance human T-Cell immunity. But they don’t say why. And Omicron’s receptor binding motif (RBM) is much more different from wild-type SARS-Cov-2.
 
“after monovalent vaccine administration, SARSCoV2 Omicron might challenge a human´s post-immunized waning antibody / B-cell responses to induce a more general and long lasting immunity by extending protective antibody repertoires and by simultaneously enhancing T-cell mediated immunity, thereby ultimately preparing an individual to defeat more pathogenic virus variants in the future. The newly reported omicron VOC (variant of concern) carries ten exchanged amino acids in its RBM of which five (K440, S446, N447, K478, and A484) are
also found in SARS-CoV1-, in bat-, and/or in civet-derived RBMs at the respective positions and through which omicron´s RBM can be distinguished from that of SARS-CoV2´s wild type”, in the article ‘Compared with SARS-CoV2 wild type´s spike protein, the SARS-CoV2 Omicron´s receptor binding motif (RBM) has adopted a more SARS-CoV1 and/or bat/civet-like
structure’,  by Michael O. Glocker, Proteome Center Rostock, University Medicine Rostock and University of Rostock, Germany, Kwabena Opuni, Hans-Juergen Thiesen, 
 
On the other hand, the Nference study group below found an EPE amino acid insertion at 214 of Omicron’s spike gene, that maps to a known human T-cell epitote. 

“We highlight that Omicron’s Spike protein harbors an insertion mutation ins214EPE that is absent in all other SARS-CoV-2 lineages……The EPE insertion on Omicron maps to the N-terminal domain (NTD) distal from the antibody binding supersite.  However, the loop where the insertion is present maps to a known human T-cell epitope on SARS-CoV-2.  Further studies will be necessary to understand whether this insertion may help SARS-CoV-2 escape T-cell immunity……the evolution of the unique insertion in Omicron could have been based on template switching during viral co-infections, or from prevalent templates in the human genome.”, ——-from the article, “Omicron variant of SARS-CoV-2 harbors a unique insertion
mutation of putative viral or human genomic origin”,
A.J. Venkatakrishnan,  and many others, nference, Cambridge, Mass.
 
So Omicron may have T-cell implications that have yet to be tested. Regarding the origin on Omicron, the Nference Group states probably a co-infection in a person, and template switching was involved.
 
The popular public opinion blames non-vaccinated people. The Michigan State study group below indicates Omicron may have been induced by vaccination or natural selection.
 
“The 32 amino acid changes, including three small deletions and one small insertion in the spike protein, suggest that Omicron may be induced by vaccination. As a result, these mutations may dramatically enhance the variant’s ability to evade current vaccines…. This result indicates that Omicron appears to have followed the infectivity-strengthening pathway of natural selection.”, from the article,  “Omicron (B.1.1.529): Infectivity, vaccine breakthrough, and antibody resistance”, by Jiahui Chen, Rui Wang, and others, Michigan State University 
 
 So Omicron may have been induced by natural selection or vaccinations according to this Michigan State study group. 
                                                                                      
Since November 2021, the rise in Chinese studies on Covid-19 origin, has been very apparent. Jinping has partially lifted his censorship of such studies. 
One study from the Chinese Academy of Sciences posits that Omicron originated in mouse cellular environment.
 
“Here, we identified 45 point mutations that Omicron acquired since divergence from the B.1.1 lineage. We found that the Omicron spike protein sequence was subjected to stronger positive selection than that of any reported SARS19 CoV-2 variants known to evolve persistently in human hosts, suggesting the possibility of host-jumping. The molecular spectrum (i.e., the relative frequency of the twelve types of base substitutions) of mutations acquired by the progenitor of Omicron was significantly different from the spectrum for viruses that evolved in human patients, but was highly consistent with spectra associated with evolution in a mouse cellular environment.”, in the article, ‘Evidence for a mouse origin of the SARS-CoV-2 Omicron variant’, Changshuo Wei, Ke-Jia Shan, and others, State Key Laboratory of Plant Genomics, Institute of Genetics and Developmental, University of Chinese Academy of Sciences, Beijing, and others.
 
Well researched paper, and well worth reading, if believable. 
                                                                                                                          
 
 Keep up with the new studies on the biorix and lancet websites. If nothing more, just read the conclusions/summaries in  the articles, and what scientific methods were used. A mind is a terrible thing to waste.  
 
 
See my other posting at  https://americaoutloud.news/strengthening-the-biological-weapons-convention/
 

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